COMPUTATIONAL ANALYSIS OF MUTATIONS IN REALLY INTERESTING NEW GENE FINGER DOMAIN AND BRCA1 C TERMINUS DOMAIN OF BREAST CANCER SUSCEPTIBILITY GENE

Authors

  • KESAVAN SABITHA Department Of Molecular OncologyCancer Institute (WIA)Sardar Patel RoadAdyarChennai
  • Ahmad Kodous Radiation Biology Department, National Center for Radiation Research & Technology, Egyptian Atomic Energy Authority, 3 Ahmed El-Zomoor st., Nasr City, Cairo, Egypt
  • Thangarajan Rajkumar Department Of Molecular Oncology Cancer Institute (WIA) Sardar Patel Road Adyar Chennai

Abstract

Objective: Breast Cancer 1 (BRCA1), Early Onset and Breast Cancer 2, Early Onset (BRCA2) genes are involved in pathways important for DNA damage
recognition, double-strand break repair, checkpoint control, transcription regulation, and chromatin remodeling. These functions are essential and
important for all cell types. Germline mutations in these genes increase the risk of breast and ovarian cancer in women. In this study, we did an
analysis of the functional and structural impact of all known single nucleotide polymorphisms (SNPs) in BRCA1 and BRCA2 using publicly available
computational prediction tools.
Methods: We analyzed the mutations using two mutation tolerance prediction approaches: Sorting intolerant from tolerant (SIFT), and polymorphism
phenotyping (PolyPhen-2). In addition, stability of the protein was analyzed by I-Mutant. Affinity and stability of really interesting new gene (RING)
and BRCA1 C-terminus (BRCT) domains were also analyzed by BioLuminate tool.
Results: Out of 486 SNPs in BRCA retrieved from functional SNP, a total of 10 SNPs were found to be deleterious by SIFT and PolyPhen. I-Mutant results
indicate that C27F, A1708V could increase the stability of protein, whereas other mutations decrease the stability. Predicted changes in stability and
affinity of RING and BRCT domains of BRCA were computed using residue scanning functionality in bioluminate for all 10 SNPs. The mutation C61R
could affect the stability of RING domain and all mutations in BRCT domain were affecting the inter subunit affinity and stability of the complex.
Conclusion: The combination of computational methods provides a way in understanding the impact of deleterious mutations in altering the BRCA
protein stability and affinity. Based on our investigation, we report potential candidate SNPs for future studies of BRCA mutations.
Keywords: Breast cancer susceptibility gene, BRCA1 C terminus domain, Zinc finger domain, Sorting intolerant from tolerant, Polymorphism
phenotyping, I-mutant, BioLuminate.

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Author Biographies

KESAVAN SABITHA, Department Of Molecular OncologyCancer Institute (WIA)Sardar Patel RoadAdyarChennai

Dr.K.Sabitha
Associate Professor
Department of Molecular Oncology

Dr Krishnamoorthy Campus

36 Sardar Patel Road
Cancer Institute (WIA)
 
Chennai 600 036
mail : rk.sabitha@gmail.com
Mobile :09840509154

Thangarajan Rajkumar, Department Of Molecular Oncology Cancer Institute (WIA) Sardar Patel Road Adyar Chennai

Professor and Head

Department Of Molecular Oncology
Cancer Institute (WIA)
Sardar Patel Road
Adyar
Chennai

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Published

01-05-2016

How to Cite

SABITHA, K. ., A. Kodous, and T. Rajkumar. “COMPUTATIONAL ANALYSIS OF MUTATIONS IN REALLY INTERESTING NEW GENE FINGER DOMAIN AND BRCA1 C TERMINUS DOMAIN OF BREAST CANCER SUSCEPTIBILITY GENE”. Asian Journal of Pharmaceutical and Clinical Research, vol. 9, no. 3, May 2016, pp. 96-102, https://journals.innovareacademics.in/index.php/ajpcr/article/view/10319.

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